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The combination of PRMT5 inhibitors and interstrand crosslinking agents induced an increase in unrepaired inter-strand crosslinks and lead to greater genomic instability (48), implying that MAT2A or PRMT5 inhibitors create a deficiency in the HR DNA repair pathway
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Likewise, after a brain injection of live Salmonella typhimurium (a bacterium implicated in food poisoning), mice engineered to overproduce human beta-amyloid displayed a lower pathogen load in the brain (Kumar et al., 2016)
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