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IGF-1 LR3 combines the best of both approaches: an N-terminal extension of 13 arginine-rich residues for steric IGFBP hindrance, Arg3 substitution to definitively abolish BP3 and BP5 binding, and retention of the 70 IGF-1 residues to maintain IGF-1R recognition

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Greenberg and co-workers (145) showed that administration of E2 reduces adipocyte size in ovariectomized female mice by reducing fatty acid uptake (down-regulation of lipoprotein lipase) and lipogenesis (down-regulation of acetyl-coenzyme A carboxylate and fatty acid synthase [FAS])