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321,336 Altered phosphorylation of insulin receptor substrate proteins, particularly IRS-1 and IRS-2, is implicated in neuronal insulin resistance, 20,337 which impairs insulin’s neurotrophic effects, leading to reduced neurite outgrowth and increased vulnerability to injury

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Another important question in the biology of NAD and CD38 is related with the potential role of the CD38 gene homologous BST-1 in organismal NAD homeostasis