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Mutagenesis experiments conducted on clathrin binding sites revealed a consistent preference of Ent1 for the Clathrin box, with the Arrestin and W-boxes appearing to play a less critical role for Ent1, serving as secondary sites of interaction

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A wide range of coupling chemistries, involving primary amines, sulfhydryls, aldehydes, hydroxyls and carboxylic acids are available for covalently attaching ligands to the matrices [12]